DYSARTHRIA In CHILDREN
- Dysarthria is a Motor Speech disorder in which the muscles that are used to produce speech are damaged, paralyzed or weakened.
The person with Dysarthria can not control tongue or voice box and may slur words
- Common causes of Dysarthria include – Nervous system disorders & conditions that cause Facial Paralysis & Tongue or Throat Muscle weakness.
- Difficult or unclear articulation of speech that is otherwise linguistically normal.
- Dysarthria is a speech disorder caused by muscle weakness. People may have trouble understanding what you say.
Speech-language pathologist or SLPs can help.
Dysarthria: Speech that is characteristically slurred, slow, and difficult to understand. A person with Dysarthria may also have problems controlling the pitch, loudness, rhythm, and voice qualities of his or her speech. Dysarthria is caused by Paralysis, Weakness, or Inability to Coordinate the Muscles of the Mouth. Dysarthria can occur as a Developmental Disability. It may be a sign of a Neuromuscular disorder such as Cerebral Palsy or Parkinson's disease. It may also be caused by a Stroke, Brain injury, or Brain tumor. Treatment of dysarthria includes intensive Speech Therapy with a focus on Oral-Motor Skill Development.
Ø Types of
Dysarthria –
> Spastic
> Flaccid
> Ataxic
> Hyperkinetic
> Hypokinetic
> Mixed
1.
Spastic
Dysarthria
-
Harsh
vocal quality
-
Reduced
Stress
-
Monopitch
Bilateral damage to the pyramidal & extrapyramidal systems.
Common causes include – Stroke, ALS, Traumatic Head Injury & MS
2.
Flaccid
Dysarthria
-
Hypernasality
-
Breathy
Voice
-
Imprecise
Consonants
Damage to the Lower Motor Neurons used in speech.
Common causes include – Physical Trauma, Brainstem Stroke, Myasthenia Gravis & Polio
3.
Ataxic
Dysarthria
-
Regular
articulatory breakdown
-
Imprecise
consonant production
Damage to the Cerebellum or the Cerebellar control circuits.
Common causes include – Degenerative disease, Stroke, Toxic
conduction, Traumatic head injury
4.
Hyperkinetic
Dysarthria
-
Depending
on associated movement disorder
-
Typically
involuntary movements
It may cases, damage to the Basal Ganglia; however some disorders
have no known causes.
Common causes include – Chorea, myoclonus, Ties, Essential tremor & Dystonia
5.
Hypokinetic
Dysarthria
-
Tremor
-
Muscular
rigidity
-
Akinesia
-
Postal
reflex disturbances
Damage to Basal Ganglia.
Common causes include – Parkinson’s disease, Neuroleptic-induced Parkinsonism, Traumatic head injury & Stroke
Ø Aphasia v/s Dysarthria –
Language Disorder
Aphasia Understanding-Expansion Problem
No
Muscle Weakness
Speech
Disorder
Dysarthria Muscular
Weakness
Language
Not Affected
Ø Causes
of Dysarthria –
1.
Developmental
Causes
-
Cerebral
Palsy
-
Down Syndrome
2.
Acquired
Causes
-
Brain
injury
-
Tumor
-
Stroke
-
Infection
Diseases
-
Atrophy
-
Toxic/Metabolism
Diseases
Dysarthria occurs when damage to the nervous system
weakens the muscles that produce speech sounds. It may affect the muscles in
one or more of the following areas:
- Face
- Lips
- Tongue
- Throat
- Upper Respiratory Tract
The neurological damage that causes dysarthria can
occur due to:
- Neurological conditions, such as Epilepsy, Amyotrophic
Lateral Sclerosis (ALS), and Parkinson’s Disease
- Brain Tumors
- Trauma from injuries to the head or neck, as well as repeated blunt
force impacts to the skull
- Inflammatory conditions, such as autoimmune
diseases, encephalitis, and meningitis
- Vascular conditions, such as stroke or Moya-Moya disease
- Exposure to toxic substances, such as alcohol, heavy metals, or carbon monoxide
Ø Sign
& Symptoms of Dysarthria –
According to the American
Speech-Language-Hearing Association, dysarthria can affect one or more of
the following five systems that speech involves:
- Respiration: Respiration moves air across the vocal cords, creating sounds that the mouth and nose shape into words.
- Phonation: This system uses airflow from the lungs plus vocal cord vibrations to produce speech sounds.
- Resonance: Resonance refers to the quality of speech sounds that the vocal tract produces.
- Articulation: This term means shaping sounds into recognizable words, which involves forming precise and accurate vowels and consonants.
- Prosody: The rhythm and
intonation of speech that give words and phrases their meaning.
The five speech systems work together, meaning that impairment in one system can affect the others.
People who have dysarthria may experience one or
more of the following symptoms:
- Abnormally quiet or loud speaking voice
- Monotonous tone
- Rough, scratchy, or hoarse voice
- Stuffy or nasal-sounding voice
- Vocal tremors
- Speech that is too fast or too slow
- Distorted consonant and vowel sounds
As conditions that cause dysarthria also affect the
nerves that control muscles, people with dysarthria may experience physical
symptoms, such as:
- Tremors or involuntary movements of the jaw, tongue, or lip
- Overly sensitive or under-sensitive gag reflex
- Muscle wasting
- Weakness
Ø Screening
of Dysarthria –
Screening for dysarthria is
pass/fail. It does not provide a diagnosis, or a detailed description of the
severity and characteristics of speech deficits associated with dysarthria but,
rather, identifies the need for further assessment.
Screening may result in
recommendations for
- dysarthria
assessment and/or
- referral for other examinations or services.
Ø Dysarthria Assessment –
Assessment of individuals with suspected
dysarthria should be conducted by an SLP using both standardized and
non-standardized measures.
The goal of the dysarthria assessment is
to
- Describe perceptual characteristics
of the individual's speech and relevant physiologic findings.
- Describe speech subsystems affected
(i.e., articulation, phonation, respiration, resonance, and prosody) and
the severity of impairment for each.
- Identify other systems and processes
that may be affected (e.g., swallowing, language, cognition); and
- Assess the impact of the dysarthria
on speech intelligibility and naturalness, communicative efficiency and
effectiveness, and participation.
The severity of the disorder does not
necessarily determine the degree of disability. Speech-related disability will
depend on the communication needs of the individual and the comprehensibility
of his or her speech in salient contexts.
Consistent with the World Health
Organization's (WHO) International Classification of Functioning, Disability,
and Health (ICF) framework (ASHA, 2016b; WHO, 2001), the assessment identifies
and describes
- Impairments in Body Structure & Function, including underlying strengths and
weaknesses in speech production and verbal/nonverbal communication.
- The individual's Limitations in Activity & Participation, including
functional status in communication, interpersonal interactions, self-care,
and learning.
- Contextual (Environmental & Personal) Factors that serve as barriers to, or facilitators
of, successful communication and life participation; and
- The impact of communication impairments on Quality of Life and functional limitations relative to the individual's premorbid social roles and abilities and the impact on his or her community.
Ø Typical
Components of The Dysarthria Assessment
The assessment process includes
consideration of the individual's hearing and vision status. This may include
hearing screening, inspection of hearing aids, and provision of an
amplification device, if needed. If the individual wears corrective lenses,
these should be worn during the assessment.
The assessment section below is not prescriptive—it
outlines the components of a very thorough exam. Some components may not be
applicable in all clinical settings.
1.
Case History
- Medical diagnosis and history
- Onset and course of symptoms
- Associated deficits (e.g.,
language, cognitive-communication, and swallowing, problems)
- Medical procedures,
hospitalizations, prior treatments and their outcomes
- Other medical and rehabilitation
specialty referrals and interventions and their outcomes
- Medications and potential side
effects/symptoms
- Review of auditory, visual, motor,
cognitive, language, and emotional status (if not included as part of the
assessment)
- Education, vocation, and cultural
and linguistic backgrounds
- Patient and family report
- Awareness, observations, and
perspectives
- Person-specific communication needs
- Impact of the presenting problem on
activities and participation
- Identification of facilitators of
and barriers to communication
- Extent to which the level of effort
for speaking changes in different contexts (e.g., when fatigued, at
different times of day, relative to medication schedule)
- Adaptability in different communication contexts (e.g., in noisy environments, with distractions, with multiple communication partners, with unfamiliar listeners)
2.
Nonspeech Examination
- Assessment of speed, strength,
range, accuracy, coordination, and steadiness of nonspeech movements and
assessment of the speech subsystems using objective measures, as
available. The following are typically included:
- Completion of a cranial nerve exam
(CN V, VII, IX, X, XI, XII)—to assess facial, oral, velopharyngeal, and
laryngeal function and symmetry
- Observation of facial and neck
muscle tone—at rest and during nonspeech activities
- Assessment of sustained vowel
prolongation—to determine if there is adequate pulmonary support and
sufficient laryngeal valving for phonation
- Assessment of alternating motion rates (AMRs) and sequential motion rates (SMRs) or diadochokinetic rates—to judge speed and regularity of jaw, lip, and tongue movement and, to a lesser extent, articulatory precision
3.
Speech Production
- Vocal quality and ability to change
loudness and pitch—to assess laryngeal/phonatory function.
- Stress testing—2 to 4 minutes of
reading or speaking aloud to assess deterioration over time (can use spontaneous
conversation, reading text aloud, or counting)
- Motor speech planning or
programming—repetition of simple and complex multisyllabic words and
sentences to determine if apraxia of speech (AOS) is present.
Prosody—use of variations in pitch, loudness, and duration to convey
emotion, emphasis, and linguistic information (e.g., meaning, sentence type,
syntactic boundaries); speech naturalness reflects prosodic adequacy
- Recommendations for speech sampling
include the following:
- Use connected speech (reading and
spontaneous speech) to observe variations in pitch, loudness, and
duration.
- Use targeted prosodic tasks,
including asking and answering questions; contrastive stress tasks; and
reading statements using prosodic variation to express different emotions.
Speech Intelligibility—the degree to which the listener (familiar/unfamiliar) understands
the individual's speech; typically reported as a percentage of words correctly
identified by a listener
- Recommendations for speech sampling
include the following:
- Use material unknown to the
listener and with low semantic predictability.
- Include words that provide a
sampling of most of the phonemes.
- Tasks include single-word
production and sentence production (recorded and later transcribed by a
judge).
Comprehensibility—the degree to which the listener understands the spoken message,
given other information or cues (e.g., topic, semantic context, gestures) to
enhance communication; typically reported as percentage of words correctly
identified by a listener
- Materials and tasks are similar to
those used to assess speech intelligibility.
- Various cues (e.g., auditory,
visual, contextual) are provided to the listener.
- The speaker's use of
comprehensibility strategies or the potential to adopt these strategies can
also be assessed during these tasks.
Efficiency—the rate at which intelligible or comprehensible speech is
communicated; typically reported as the number of intelligible or
comprehensible words per minute
- Sentence-level materials and tasks
are similar to those used to assess speech intelligibility and
comprehensibility.
- Sentences are transcribed by a
judge, and the number of correct words per minute are computed.
Other components of the assessment may
include a review of the following, which may lead to further, in-depth
assessment of these areas:
- Language—assess
receptive and expressive language skills in oral and written modalities to
help distinguish between dysarthria and aphasia.
- Cognitive-Communication—identify aspects of verbal or nonverbal
communication that may be affected by disruptions in cognition (e.g.,
attention, memory, organization, executive function).
- Swallowing—assess swallowing function.
Assessment May Result in The Following Outcomes:
- Diagnosis of dysarthria and
classification of dysarthria type.
- Clinical description of the dominant
auditory-perceptual speech characteristics and the severity of the
disorder.
- Presence of co-morbid conditions,
including apraxia of speech, aphasia, cognitive-communication disorder, or
swallowing disorder.
- Statement of prognosis and
recommendations for intervention that relate to overall communication
adequacy.
- Development of an intervention or
management plan (in collaboration with patient, family, and rehabilitation
team), including (a) prosthetic or surgical management or (b) augmentative
and alternative communication (AAC), as appropriate.
- Identification of relevant follow-up
services, including support for individuals with dysarthria.
- Referral to other professionals as needed (e.g., neurologist, psychologist).
Ø Treatment of Dysarthria –
v Treatment Approaches
Treatment can be restorative (i.e.,
aimed at improving or restoring impaired function) and/or compensatory (i.e.,
aimed at compensating for deficits not amenable to retraining).
·
Restorative approaches focus on improving
·
Speech intelligibility,
·
Prosody and naturalness, and
·
Efficiency.
·
Compensatory approaches focus on
·
Improving comprehensibility by
o increasing the speaker's use of communication strategies,
o improving listener skills and capacity, and
o altering the communication environment
·
Increasing effective use of AAC options;
and
·
Increasing use of non-AAC devices
Ø Treatments That Target Speech-Production Subsystems
· Respiration
- Making postural adjustments (e.g.,
sitting upright to improve breath support for speech)
- Inhaling deeply before onset of
speech utterance (known as preparatory
inhalation)
- Using optimal breath groups when
speaking (i.e., for each breath, speak only the number of syllables that
can be comfortably produced)
- Using expiratory muscle strength
training to improve strength of the expiratory muscles (the individual
blows into a pressure threshold device with enough effort to overcome a
preset threshold)
- Using inspiratory muscle strength
training to improve strength of the inspiratory muscles to permit better
sustained or repeated inspirations (the individual uses a handheld device
that is set to require a minimum inspiratory pressure for inspiration to
continue)
- Using maximum vowel prolongation
tasks to improve duration and loudness of speech
- Using controlled exhalation tasks
(air is exhaled slowly over time) to improve control of exhalation for
speech
- Using nonspeech tasks to improve subglottal air pressure and respiratory support (e.g., blowing into a water glass manometer)
·
Phonation
- Lee Silverman Voice Treatment
- Pitch Limiting Voice Treatment
- Effort closure techniques to
increase adductory forces of vocal folds (e.g., pulling upward on chair
seat; squeezing palms of hands together)
- Improved timing of phonation (e.g., initiating phonation at beginning of expiration)
·
Articulation
- Phonetic placement techniques (e.g.,
hands-on, descriptive, pictures) to work on positioning of the mouth,
tongue, lips, or jaw during speech.
- Phonetic derivation techniques
(nonspeech to speech tasks such as "blowing" to /u/).
- Exaggerated articulation (over-articulation)
to emphasize phonetic placement and increase precision, sometimes called
"clear speech."
- Minimal contrasts to emphasize sound
contrasts necessary to differentiate one phoneme from another.
- Intelligibility drills in which the
individual reads words, phrases, or sentences and attempts to repair
content not understood by the listener.
- Rate modification to facilitate
articulatory precision—strategies include
- pausing at natural linguistic
boundaries (e.g., using printed script marked at natural pauses);
- using external pacing methods such
as pacing boards, hand/finger tapping, and alphabet boards;
- using auditory feedback (e.g.,
delayed auditory feedback or metronome);
- using visual feedback (e.g., using
computerized voice programs); and
- using approaches that reduce speech rate without directly targeting it (e.g., increasing loudness, altering pitch variation, altering phrasing or breath patterns).
·
Resonance
- Prosthetic management in collaboration
with other disciplines (e.g., dentists, prosthodontists)—examples include
- palatal lift prosthesis and
- nasal obturator to occlude nasal
airflow.
- Resistance training during speech using continuous positive air pressure.
·
Prosody
- Increasing awareness and ability to
control respiration, rate, and pitch to vary emphasis within multisyllabic
words and in connected utterances (e.g., using scripts, marked and
unmarked passages)
- Improving intonation by signaling
stress with loudness, pitch, or duration
- Extending breath groups to better
align with syntactic boundaries
- Using contrastive stress tasks to improve prosody and naturalness (e.g., repeating sentence with stress on different word[s])
Other Treatment Options
Ø Communication Strategies
A variety of communication strategies can
be used by the individual with dysarthria (speaker) and his or her
communication partner to enhance communication when speech intelligibility or
efficiency is reduced. These strategies can be used before, during, or after
other treatment approaches are implemented to improve or compensate for speech
deficits (see, e.g., Duffy, 2013).
Speaker Strategies include
- maintaining eye contact with the
communication partner;
- preparing the communication partner
by gaining his or her attention and introducing the topic of conversation
before speaking;
- pointing and gesturing to help
convey meaning;
- looking for signs that the
communication partner has or has not understood the message; and
- effectively using conversational repair strategies (e.g., restating message in different words; using gestures to help clarify message).
Ø Communication-partner Strategies include
- maintaining eye contact with the
speaker;
- being an active listener and making
every effort to understand the speaker's message;
- asking for clarification by asking
specific questions;
- providing feedback and
encouragement; and
- optimizing the ability to hear the speaker and to see the speaker's visual communication cues (e.g., by wearing prescribed hearing aids and glasses during conversations).
Ø Environmental Modification
Environmental modification involves identifying optimal parameters to enhance
comprehensibility.
These parameters include
- reducing background noise (e.g.,
choose a quiet setting for conversations; turn off TV, radio, and fans);
- ensuring that the environment has
good lighting
- improving proximity between the
speaker and his or her communication partner; and
- using face-to-face seating for
conversations.
Augmentative
and Alternative Communication (AAC)
AAC involves
supplementing or replacing natural speech and/or writing.
The two forms of AAC are
- unaided (e.g., manual signs,
gestures, and finger spelling) and
- aided (e.g., line drawings,
pictures, communication boards, tangible objects, speech-generating
devices).
Other augmentative supports include voice amplifiers, artificial phonation devices (e.g., electrolarynx devices and intraoral devices), and oral prosthetics to reduce hypernasality.
Ø Medical/Surgical Intervention
SLPs may refer the individual to a
medical specialist to assess the appropriateness of, or need for, medical
interventions.
These interventions can include, for
example,
- pharyngeal augmentation, pharyngeal
flap, or palatal flap to treat velopharyngeal incompetency and improve
resonance
- laryngeal (vocal fold) augmentation
(e.g., autologous fat or collagen), laryngoplasty, or recurrent laryngeal
nerve sectioning to improve phonation; and
- pharmacological management to
relieve symptoms of the underlying neurologic condition (e.g., spasticity,
tremor) associated with underlying neurologic disease.